Unlike cancer, many diseases appear to be dormant in the population with a more acceptable cloak of invisibility, with controllability and a relatively low risk of death. Parents often talk about the antihypertensive drugs and lipid-lowering drugs that are always available around us. They all aim at a disease caused by excessive production of bad fat in the body and the formation of embolism in the cardiovascular and cerebrovascular vessels. Long-term use of such drugs has become the living habit of many patients. Medicines are in hand, and patients’ quality of life is locked in tiny capsules and tablets. Long-term medication has become a norm, even if it brings many inconveniences. But in the not-so-distant future, this inconvenience may be overcome by gene editing. On July 13, a volunteer in New Zealand has become the first person to undergo DNA editing to lower blood cholesterol, a step that could herald the technology’s widespread use to prevent heart attacks. The experiment, part of a clinical trial by U.S. biotech company Verve Therapeutics, uses injections of the gene-editing tool CRISPR to modify the bases of DNA in patients’ liver cells. According to the company, this tiny edit is enough to permanently lower a person’s &ld .
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